Cancer metabolism is characterized by a series of biochemical changes that allow malignant cells to sustain rapid growth and proliferation. Central to these changes are metabolites, which serve as both substrates and signaling molecules that drive tumour igenesis. This article explores the role of metabolites in cancer metabolism, focusing on key pathways such as glycolysis, the tricarboxylic acid (TCA) cycle, and the Pentose Phosphate Pathway (PPP). We also discuss the implications of metabolite alterations in tumour igenesis, their potential as biomarkers, and therapeutic targets. Understanding these metabolic adaptations offers insight into cancer biology and highlights opportunities for novel treatment strategies.
